By J. Walden, H. Grunze

ISBN-10: 3805571755

ISBN-13: 9783805571753

Complement factor: Neuropsychobiology 2000, Vol. forty two, Suppl. 1

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Extra info for Bipolar Illnesses: New Ways of Treatment (Stanley Symposium, Munich, October 1999 (Neuropsychobiology))

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S. s. s. s. 03 Neuropsychobiology 2000;42(suppl 1):33–37 Method EEG was obtained from 12 patients with affective disorders (ICD-10: F31, F32, F33) of the Outpatient Clinic for Relapse Prophylaxis in Affective Disorders who were included in a prospective study. 5 months after baseline under lithium (T2). The patients had analyzable recordings for quantitative EEG analysis at both time points. The age of the patients (5 females, 7 males) ranged from 38 to 76 years (mean 59 B 13 years). Before lithium treatment (T1), 3 patients were treated with neuroleptics and antidepressants, 6 patients had antidepressive treatments without other psychotropic drugs, and 3 patients were not treated with any psychotropic medication.

2c, 3c) and acceleration (fig. 2d, 3d) over time. In particular the graph of performance on a manic day (fig. e. number of local maxima and minima, between each pair of the dividing vertical lines. The high number of inversions reflects the poor automatization on that day, as compared to the fewer number of inversions between dividing lines on the depressed day (fig. 3d). After 4 weeks treatment with valproate, the periodic change in the patterns of the automatization parameters of handwriting disappeared concurrently with the stabilization of clinical and self-ratings (fig.

A concentration of 10 ÌM LTG in our experimental setting corresponds to estimated therapeutic plasma levels of about 4–16 ÌM in humans, especially if 55% plasma protein binding is taken into account [9]. We have found effects of LTG on neurotransmitter release only for concentrations far above the pharmacologically relevant ones. However, at these concentrations, effects of LTG on population spikes were much stronger than on fEPSP. This is in contradiction to previous reports and may be due to higher concentrations or a pharmacologically induced transmitter release in those studies [5, 10, 11].

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Bipolar Illnesses: New Ways of Treatment (Stanley Symposium, Munich, October 1999 (Neuropsychobiology)) by J. Walden, H. Grunze


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